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Russell-silver Syndrome: Disease Bioinformatics

Research of Russell-silver Syndrome has been linked to Dwarfism, Growth Disorders, Fetal Growth Retardation, Uniparental Disomy, Growth Retardation. The study of Russell-silver Syndrome has been mentioned in research publications which can be found using our bioinformatics tool below. Researched pathways related to Russell-silver Syndrome include Methylation, Dna Methylation, Secretion, Pathogenesis, Demethylation. These pathways complement our catalog of research reagents for the study of Russell-silver Syndrome including antibodies and ELISA kits against GROWTH HORMONE, CDKN1C, EGFR, GH1, GRB10.

Top Research Reagents

We have 3339 products for the study of Russell-silver Syndrome that can be applied to Flow Cytometry, Immunocytochemistry/ Immunofluorescence, Immunohistochemistry, Western Blot from our catalog of antibodies and ELISA kits.

NBP3-42997
Immunohistochemistry: MEST Antibody (5E0) [NBP3-42997] - Immunohistochemistry of paraffin-embedded human prostate cancer tissue slide using (MEST Antibody) at dilution of 1:400Western Blot: MEST Antibody (5E0) [NBP3-42997] - PC-3 cells were subjected to SDS PAGE followed by western blot with (MEST antibody) at dilution of 1:1000

Mouse Monoclonal
Species Human, Mouse
Applications WB, ELISA, ICC/IF

NBP1-89917
Immunohistochemistry-Paraffin: p57 Kip2 Antibody [NBP1-89917] - Staining in human placenta and colon tissues using anti-CDKN1C antibody. Corresponding CDKN1C RNA-seq data are presented for the same tissues.Western Blot: p57 Kip2 Antibody [NBP1-89917] - Analysis in human placenta tissue.

Rabbit Polyclonal
Species Human
Applications WB, ICC/IF, IHC

1 Publication

Related Genes

Russell-silver Syndrome has been researched against:
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Related PTMs

Russell-silver Syndrome has been studied in relation to posttranslational modifications (PTMs) including:

Alternate Names

Russell-silver Syndrome is also known as Russell Silver Syndrome, Russell's Syndrome, Silver Russell Syndrome, Silver Syndrome, Silver-russell Dwarfism.